KMID : 0608619930030010079
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Korean Journal of Aerospace and Environmental Medicine 1993 Volume.3 No. 1 p.79 ~ p.88
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An Experimental Study on the Change of EKG in Hyperbaric Oxygenation
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Abstract
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To estimate the effects of hyperbaric oxygenation on electrocardiographic changes and to estimate the role of hypoxia induced by carbon monoxide poisoning in hyperbaric oxygenation, 20 rabbits were continuously exposed to 4 different conditions ;
1)
breathing ambient air for 30min. followed by 100%-1ATA. Oxygen (AA-NBO group), 2) and by 100%-4ATA. Oxygen (AA-HBO group), 3) breathing carbon monoxide (4,000ppm) for 30 min. followed by 100%-1ATA. Oxygen (CO-NBO group), 4) and by 100%-4ATA.
oxygen
(CO-HBO group). The EKG, the time to dyspnea and convulsion during exposure were recorded and analyzed
Dyspnea was observed in RA-HBO and CO-HBO groups only and the occurrence rates were 80%, 100%, respectively. Convulsion and abnormal EKG findings were observed also in above groups only and the convulsion rates were 20% in both groups and the
rate
of
abnormal EKG findings were 80% in both groups. Death was observed in 2 cases of CO-HBOP group.
Abnormal EKG findings observed were Sinus-bradycardia, T-inversion and ST-T changes (St depression and elevation) in the order and frequency of appearances (p>0.05). Those changes showed a tendency to appear earlier in CO-HBO group than in RA-HBO
group
(p>0.05) when compad by the time of exposure, abnormal EKG findings and dyspnea appeared earlier than dyspnea (p>0.05), former two didn't show any difference in the time of onset.
These results suggest that the principal hazard of hyperoxia of EKG is myocardial ischemia involving ST-T changes and arrhythmia, and that the preceding hypoxic condition may exaggerate the following hyperoxic injury.
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KEYWORD
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